A 69-year-old man had new sustained ventricular tachycardia caused by flecainide which promptly responded to intravenous lidocaine therapy. Discontinuation of the lidocaine infusion resulted in the reappearance of ventricular tachycardia which again immediately terminated after lidocaine was given. In this case, lidocaine effectively reversed the proarrhythmic effects of flecainide.
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Many drugs may induce QT interval prolongation on ECG and lead to this syndrome. ... Ventricular tachycardia treatment Treatment and cure for paroxysmal ventricular ...
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Ablation for Ventricular Tachycardia What is an ablation [Ablation (sounds like ah-blay-shun) is a medical procedure used to correct certain heart rhythm problems, such as ventricular tachycardia (VT). It restores the normal regular rhythm of the heart by scarring the tissue in the heart that triggers the abnormal rhythm.
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Certain medications have been known to trigger the pounding heart rhythm of SVT. These include: Digoxin ( Digitek, Digox, Lanoxin ), for treating heart failure. Theophylline ( Elixophyllin ...
Answer: Atropine Question 26 A 12-lead ECG shows ventricular tachycardia in a stable patient with a pulse. Which of the following medications could be considered for this patient? Answer: Amiodarone, procainamide or sotalol Question 27 A patient is in ventricular fibrillation. After two shocks, the rhythm remains the same.
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Patients already taking these drugs may present with rates of around 200, and sometimes the AV node then allows 1:1 transmission, resulting in a ventricular rate of 200 or so. Cardinal Features: Classic sawtooth Flutter waves may be seen best in 11, 111, AVF and V1 (see Figure 13).
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The mainstays of long-term treatment for clinically stable patients with ventricular tachycardia are the various antiarrhythmic drugs. However, cardiologists are increasingly making use of interventional therapy with devices and ablation procedures designed to abort ventricular tachycardia or to destroy arrhythmogenic tissue in the heart.
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Mar 19, 2019 · Ventricular tachycardia (VT) is associated with increased mortality in patients with a history of MI. ICD implantation is currently the standard of care for the prevention of sudden cardiac death, and contributes to a reduction of total mortality. 1 Despite effective treatment of ventricular arrhythmias with the use of anti-tachycardia pacing (ATP) or shocks, ICDs do not prevent VT.
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Triggered by a premature ventricular contraction (R-on-T PVC) Treatment. The treatment of torsades de pointes aims to restore a normal rhythm and to prevent the arrhythmia recurring. While torsades may spontaneously revert to a normal sinus rhythm, sustained torsades requires emergency treatment to prevent cardiac arrest.
With appropriate drug or surgical treatment, ventricular tachycardia can be controlled in most people. Prevention A person susceptible to sustained ventricular tachycardia often has a small abnormal area in the ventricles that is the source of the trigger event.
Ventricular tachycardia (VT) is a rapid heartbeat that starts in the lower chambers of the heart (ventricles). An implantable cardioverter-defibrillator (ICD) is a device designed to quickly detect a life-threatening, rapid heartbeat coming from the bottom chamber of the heart.
Catecholaminergic polymorphic ventricular tachycardia (CPVT) is an increasingly recognized disorder, typically of the ryanodine receptor, characterized by exertionally triggered rapid atrial and ventricular tachycardia and ventricular fibrillation. Triggered arrhythmia may also play a role in poisoning by antiarrhythmic drugs (eg, digoxin).
Ventricular Tachycardia can be diagnosed by an electrocardiogram (ECG). Treatment Anti-arrhythmic drugs such as lidocaine, procainamide, and sotalol may need to be used initially to convert the arrhythmia in an acute episode, and may need to be continued indefinitely.